What cause acne?

Question

I want to know what causes acne. Is it true that chocolate, oil, etc. dont cause acne? I read somewhere that stress is what really causes it. LIke emotional stress or motional stress w/e it was. I used proactive and it works at times and at other times it doesnt and ive used it for like 3-4 months. Now i use Klear Action which is like proactive and it worked well in the beginning but now its like not doing anything anymore. I do have some scares now cause of before, is there a way to heal them up or get them clearer? Klear Action is working slowly on it though. I use it everyday in the morning and at night. Does it really make a difference if you dont put sunscreen on? Before when i was using Proactive i didnt put some on for a few weeks and my face clears up by itself but not entirely cause then later i get them again.

Answer 1

chocolate does not cause acne. its from you ace producing too much oil and it clogging your pores. make sure you don't over wash your face because that will cause your face to produce more oil to make up for the drying and then it will lead to pimples.

Answer 2

Dry and/or greasy skin due to poor oil composition of the skin. Thick greasy oils clog pores, lack of oil leads to dryness and irritation. You need thin oils to moisturize while dissolving and clearing gunk in your poors. Try fish oil or seafood. 2 tsp fish oil a day or 4 servings of seafood a week. Stick it out for at least 2 months; it will take a long time to replace all your oil. Any effect after 1-2 days is temporary or random; so even if it makes you break out a little at first, you haven't given it a full try yet.



In the short term you can wash and moisturize well, but that will only go so far. Plus excessive washing can be drying and excessive moisturizing can be clogging. Use a small amount of a light moisturizer, made with oil not jelly or grease. Often that means soybean oil or mineral oil. Mineral oil means mined from the ground. So soybean oil is usually better, though mineral oil won't cause too much harm. Clean with soap and water, not a harsh acne cleanser. Even then they only work so well. So you really need the seafood.



Antibiotics aren't really good for bacteria long term, they'll come back in force after. Short term they may help. After you get off them find some kefir with acidophilus listed first or 2nd to replace the friendly bacteria they destroyed. Studies show less illness when you have these bacteria, even outside the stomach in places such as the lungs and elsewhere. You want friendly bacteria to fill the void when the antibiotics stop, not harmful ones.



In the short term you might also try 100,000+ iu retinol vitamin A (a megadose, and too much for normal use) or one of the acne drugs that is similar to retinol vitamin A. It's some minor harm to your organs, but it helps against bacteria on your skin. At least it doesn't have the other long term drawbacks to your skin that antibiotics and many scrubs do.



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Answer 3

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Answer 4

The exact cause of acne is unknown, but doctors believe it results from several related factors. One important factor is rising hormone levels. These hormones, called androgens (male sex hormones), increase in both boys and girls during puberty and can cause the sebaceous glands to enlarge and make more sebum. Another factor is heredity or genetics. Researchers believe that the tendency to develop acne can be inherited from parents. For example, studies have shown that many school-age boys with acne have a family history of the disorder.



Myths About the Causes of Acne



There are many myths about what causes acne. Chocolate and greasy foods are often blamed, but research has shown that foods seem to have little effect on the development and course of acne in most people. Another common myth is that dirty skin causes acne; however, blackheads and other acne lesions are not caused by dirt.

Answer 5

Well, I don't exactly have U'r problem but about the skin yes I do. Stress cause pimple not acne but pimple can cause acne. Anyway pimple or acne still make you stress. So i think you should is get some stress therapy to get rid of the stress (i recommended Paul Mckenna Cd's, it's up to you.) Then you can used some soap free face cleaning like Avène products. That's what i did and it help. Good Luck!

Answer 6

Acne is considered an abnormal response to normal levels of the male hormone testosterone. Don't over wash your face, twice is pretty much all you need a day. Use a hypoallergenic cleanser, spot treatments that contain benzioc peroxide is pretty much all you need for mild cases. A MD might prescribe you with a dose of antibiotics and if serious, accutane.

Answer 7

Wask your face with a mild acne cleanser twice a day. Take vitamin A pills. Don't get too stessed. Try yoge or something like that. Good luck.

Answer 8

Stress/hormones do cause break outs. Chocolate doesn't unless it causes you to have a hormonal imbalance. The things that make a pimple are dead skin, oil, and bacteria. To reduce the number of bacteria, It's important to keep your face as clean as possible, and use an exfoliate to slough away dead skin to keep pimples from forming. And always use a moisturizer, so your skin doesn't get too dry. Proactive didn't really work for me, b/c it dried my cheeks out, but MD skincare alpha/beta pads saved my face! And I use Philosophy Hope in a jar as moisturizer. Good for all skin-types, non-oily

Answer 9

oil pores clogged with excess body oil and dead skin cells. Then bacteria forms in these pores, and when this happens, your body sends white blood cells to fight this bacteria. this makes the pore swell, and the white puss is just a mixture of dead white blood cells, dead skin cells and excess oil. oily skin is very prone to acne, and acne develops more in the summer, becuase there is an increase in the production of oil from the pores due to the heat.

Answer 10

Exactly why some people get acne and some do not is not fully known. It is known to be partly hereditary. Several factors are known to be linked to acne:



* Hormonal activity, such as menstrual cycles and puberty

* Stress, through increased output of hormones from the adrenal (stress) glands.

* Hyperactive sebaceous glands, secondary to the three hormone sources above.

* Accumulation of dead skin cells.

* Bacteria in the pores, to which the body becomes 'allergic'.

* Skin irritation or scratching of any sort will activate inflammation.

* Use of anabolic steroids.

* Any medication containing halogens (iodides, chlorides, bromides), lithium, barbiturates, or androgens.

* Exposure to high levels of chlorine compounds, particularly chlorinated dioxins, can cause severe, long-lasting acne, known as Chloracne.



Traditionally, attention has focused mostly on hormone-driven over-production of sebum as the main contributing factor of acne. More recently, more attention has been given to narrowing of the follicle channel as a second main contributing factor. Abnormal shedding of the cells lining the follicle, abnormal cell binding ("hyperkeratinization") within the follicle, and water retention in the skin (swelling the skin and so pressing the follicles shut) have all been put forward as mechanisms involved.



Several hormones have been linked to acne: the male hormones testosterone, dihydrotestosterone (DHT) and dehydroepiandrosterone sulfate (DHEAS), as well as insulin-like growth factor 1 (IGF-I). In addition, acne-prone skin has been shown to be insulin resistant.



Development of acne vulgaris in latter years is uncommon, although this is the age group for Rosacea which may have similar appearances. True acne vulgaris in older adults may be a feature of an underlying condition such as pregnancy and disorders such as polycystic ovary syndrome or the rare Cushing's syndrome.

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Misconceptions about causes



There are many misconceptions and rumors about what does and does not cause the condition:



* Diet. One flawed study [1] purported that chocolate, french fries, potato chips and sugar, among others, affect acne. A recent review of scientific literature cannot affirm either way [2]. The consensus among health professionals is that acne sufferers should experiment with their diets, and refrain from consuming such fare if they find such food affects the severity of their acne[3]. A recent study[4], based on a survey of 47,335 women, did find a positive epidemiological association between milk consumption and acne, particularly skimmed. The researchers hypothesize that the association may be caused by hormones (such as bovine IGF-I) present in cow milk; but this has not been definitively shown. Seafood, on the other hand, may contain relatively high levels of iodine, but probably not enough to cause an acne outbreak. Still, people who are prone to acne may want to avoid excessive consumption of foods high in iodine. It has also been suggested that there is a link between a diet high in refined sugars and acne. According to this hypothesis, the startling absence of acne in non-westernized societies could be explained by the low glycemic index of these tribes' diets. [5] Further research is necessary to establish whether a reduced consumption of high-glycemic foods (such as soft drinks, sweets, white bread) can significantly alleviate acne, though consumption of high-glycemic foods should in any case be kept to a minimum, for general health reasons. [6]

* Deficient personal hygiene. Acne is not caused by dirt. This misconception probably comes from the fact that acne involves skin infections. In fact the blockages that cause acne occur deep within the narrow follicle channel, where it is impossible to wash them away. These plugs are formed by the cells and sebum created there by the body. The bacteria involved are the same bacteria that are always present on the skin. Regular cleansing of the skin can reduce, but not prevent, acne for a particular individual and very little variation among individuals is due to hygiene. Anything beyond very gentle cleansing can actually worsen existing lesions and even encourage new ones by damaging or overdrying skin.

* Sex. Common myths state that either celibacy or masturbation cause acne and, conversely, that sexual intercourse can cure it. There is no scientific evidence suggesting that any of these are factual. It is true, though, that anger and stress affect hormone levels and thus bodily oil production, which can cause acne.

Answer 11

Clogged pores that have bacteria in them. and chocolate doesn't cause acne

Answer 12

I had this problem but after using lakhme calamine and cleansing lotion and astringent, my face cleared. Now the lakhme company has stopped producing calamine product, but lacto calamine is still available if it suits your skin. Relax, drink lots of water, exercise, avoid oily foods, maybe too much sugar also is bad (not sure about that).

Answer 13

http://www.acne.com/cause_effects/causes_acne.php

Answer 14

Several factors contribute to the development of acne. The primary problem is that the abnormal flaking of cells inside the hair follicle leads to the formation of a plug. The plug can enlarge and even rupture the hair follicle. A ruptured hair follicle spills its contents of oil and debris into the skin where it leads to swelling and causes redness (inflammation).



Bacteria that normally live on the skin also play a role in acne development. The bacteria known as Propionibacterium acnes are responsible for causing acne. These bacteria produce substances that cause redness and irritation (inflammation). They also make enzymes, which dissolve the sebum (oil from oil glands in the skin) into irritating substances. These substances also make the inflammation worse.





Certain hormones called androgens are an additional factor in causing acne. Androgens are male hormones that are present in both men and women, but are higher in men. Androgens do two things: First, they enlarge the sebaceous glands in the skin. Second, they cause these glands to increase sebum (oil) production. The increased sebum leads to plug formation and serves as more "food" for the bacteria. Androgens surge at puberty, which is why teens develop armpit and pubic hair, and why boys develop facial hair and deeper voices. This hormonal surge also contributes to the development of acne in teens.





Estrogens, which are the female hormones, actually can help to improve acne in girls. A woman's monthly menstrual cycle is due to changes in the estrogen levels in her body. This is why acne in a female may get better and then get worse as she goes through her monthly cycle. A doctor may recommend acne treatment with birth control pills, which contain the helpful estrogens.





We also now believe that acne can run in some families. This may be due to some genetic factor that has not yet been discovered.





Anatomy of the hair follicle: Hair follicles exist on virtually all skin except for the palms of the hands and soles of the feet. Inside the follicle, the hair extends up from the deep layers of the skin and comes out of a pore. Near the surface, the oil gland (sebaceous gland) enters the hair follicle where it empties oil (sebum) at a relatively constant rate. The sebum lubricates the skin and provides a protective barrier to prevent drying. Skin on the face, chest, and back has an especially large number of sebaceous glands. These are the areas where acne occurs.





Acne lesions: There are 2 major types of acne lesions: noninflammatory and inflammatory. Noninflammatory acne lesions include blackheads (open comedones) and whiteheads (closed comedones). Open and closed comedones along with papules and pustules are referred to as papulopustular acne—a form of inflammatory acne. Nodular acne is the most severe form of inflammatory acne.





Noninflammatory acne: Open comedones result from the enlargement and dilation of a plug that forms from oil and flakes of skin inside the hair follicle.





The hair follicle pore remains open exposing a black plug (known as a blackhead). The dark color is not dirt inside the pore. Instead it is the oil inside the pore, which has become exposed from the outside air.





A closed comedo forms if the hair follicle pore remains closed. The plug in a closed comedo or whitehead is therefore not exposed to the outside air, and no black color develops. The closed comedo simply appears as a tiny, sometimes pink bump in the skin.





Inflammatory acne: Inflammatory acne lesions consist of red blemishes, pimples also called zits (papules, pustules), and larger, deeper swollen tender lesions (nodules).





Papules are closed comedos, which have become red, swollen, and inflamed.





Pustules are closed comedos, which become inflamed and begin to rupture into the skin forming pustular heads of various sizes.





Nodules represent large, tender, swollen acne lesions, which have become intensely inflamed and rupture under the skin. If untreated, these can produce deep scarring









The pathogenesis of acne vulgaris is multifactorial. Four key factors are responsible for the development of an acne lesion. These factors are follicular epidermal hyperproliferation with subsequent plugging of the follicle, excess sebum, the presence and activity of Propionibacterium acnes, and inflammation.



Follicular epidermal hyperproliferation is the first recognized event in the development of acne. The exact underlying cause of this hyperproliferation is not known. Currently, the 3 leading hypotheses have been proposed to explain why the follicular epithelium is hyperproliferative in individuals with acne.



First, androgen hormones have been implicated as the initial trigger. Comedones, the clinical lesion that results from follicular plugging, begin to appear around adrenarche in persons with acne. Furthermore, the degree of comedonal acne in prepubertal girls correlates with circulating levels of the adrenal androgen dehydroepiandrosterone sulfate (DHEA-S). Additionally, androgen hormone receptors are present in the portion of the follicle where the comedone forms; individuals with malfunctioning androgen receptors do not develop acne.



Second, changes in lipid composition have been implicated in the development of acne vulgaris. Persons with acne frequently have excess sebum production and oily skin. This excess sebum may dilute the normal epidermal lipids and result in a change in the relative concentrations of the various lipids. Diminished concentrations of linoleic acid have been demonstrated in individuals with acne and, interestingly, these levels normalize after successful treatment with isotretinoin. This relative decrease in linoleic acid may be what initiates comedone formation.



Inflammation is the third hypothesized factor incriminated in comedone formation. Interleukin (IL)–1–alpha is a proinflammatory cytokine. It has been used in a tissue model to induce follicular epidermal hyperproliferation and comedone formation. Although inflammation is not apparent microscopically or clinically in early lesions of acne, it may still play a pivotal role in the development of acne vulgaris and the comedones.



Excess sebum is another key factor in the development of acne vulgaris. Sebum production and excretion are regulated by a number of different hormones and mediators. Androgen hormones, in particular, promote sebum production and release. Still, most men and women with acne have normal circulating levels of androgen hormones. An end-organ hyperresponsiveness to androgen hormones has been hypothesized. Androgen hormones are not the only regulators of the human sebaceous gland. Numerous other agents, including growth hormone and insulinlike growth factor, also regulate the sebaceous gland and may contribute to the development of acne.



P acnes is a microaerophilic organism present in many acne lesions. Although, it has not been shown to be present in the earliest lesions of acne, the microcomedo, its presence in later lesions is almost certain. The presence of P acnes promotes inflammation through a variety of mechanisms. P acnes stimulates inflammation by producing proinflammatory mediators that diffuse through the follicle wall. Recent studies have shown that P acnes binds to the toll-like receptor 2 on monocytes and neutrophils. Binding of the toll-like receptor 2 then leads to the production of multiple proinflammatory cytokines, including IL-12, IL-8, and tumor necrosis factor. Hypersensitivity to P acnes may also explain why some individuals develop inflammatory acne vulgaris while others do not.



Inflammation may be a primary phenomenon or a secondary phenomenon. Most of the evidence to date suggests a secondary inflammatory response to P acnes as mentioned above. However, IL-1-alpha expression has been identified in the microcomedone, and it may play a role in the develop

Answer 15

MASTERBATION